Hepatología: Metabolismo, Epigenética y Carcinogénesis
METACAN
University of Edinburgh
Edimburgo, Reino UnidoPublications en collaboration avec des chercheurs de University of Edinburgh (8)
2022
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Activation of the unfolded protein response (Upr) is associated with cholangiocellular injury, fibrosis and carcinogenesis in an experimental model of fibropolycystic liver disease
Cancers, Vol. 14, Núm. 1
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Clinical relevance of biomarkers in cholangiocarcinoma: critical revision and future directions
Gut, Vol. 71, Núm. 8, pp. 1669-1683
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Genetic and pharmacological inhibition of XBP1 protects against APAP hepatotoxicity through the activation of autophagy
Cell death & disease, Vol. 13, Núm. 2, pp. 143
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New molecular mechanisms in cholangiocarcinoma: signals triggering interleukin-6 production in tumor cells and KRAS co-opted epigenetic mediators driving metabolic reprogramming
Journal of Experimental and Clinical Cancer Research, Vol. 41, Núm. 1
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Next-generation sequencing of bile cell-free DNA for the early detection of patients with malignant biliary strictures
Gut, Vol. 71, Núm. 6, pp. 1141-1151
2021
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Dual Targeting of G9a and DNA Methyltransferase-1 for the Treatment of Experimental Cholangiocarcinoma
Hepatology, Vol. 73, Núm. 6, pp. 2380-2396
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Fibrotic events in the progression of cholestatic liver disease
Cells, Vol. 10, Núm. 5
2019
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Mitogen-Activated Protein Kinases (MAPKs) and Cholangiocarcinoma: The Missing Link
Cells, Vol. 8, Núm. 10