Linfomas
GLUN
Albert Einstein College of Medicine
Nueva York, Estados UnidosPublications en collaboration avec des chercheurs de Albert Einstein College of Medicine (22)
2022
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Correction to: Intraclonal Complexity in Chronic Lymphocytic Leukemia: Fractions Enriched in Recently Born/Divided and Older/Quiescent Cells (Molecular Medicine, (2011), 17, 11-12, (1374-1382), 10.2119/molmed.2011.00360)
Molecular Medicine
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Role of EXO1 nuclease activity in genome maintenance, the immune response and tumor suppression in Exo1D173Amice
Nucleic Acids Research, Vol. 50, Núm. 14, pp. 8093-8106
2020
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CLL intraclonal fractions exhibit established and recently acquired patterns of DNA methylation
Blood Advances, Vol. 4, Núm. 5, pp. 893-906
2014
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Genome-wide expression profiling of B lymphocytes reveals IL4R increase in allergic asthma
Journal of Allergy and Clinical Immunology, Vol. 134, Núm. 4, pp. 972-975
2013
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Mammalian Exo1 encodes both structural and catalytic functions that play distinct roles in essential biological processes
Proceedings of the National Academy of Sciences of the United States of America, Vol. 110, Núm. 27
2012
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AIDing antibody diversity by error-prone mismatch repair
Seminars in Immunology, Vol. 24, Núm. 4, pp. 293-300
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Germline deletion of Igh 3′ regulatory region elements hs 5, 6, 7 (hs5-7) affects B cell-specific regulation, rearrangement, and insulation of the Igh locus
Journal of Immunology, Vol. 188, Núm. 6, pp. 2556-2566
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IGHV-unmutated and IGHV-mutated chronic lymphocytic leukemia cells produce activation-induced deaminase protein with a full range of biologic functions
Blood, Vol. 120, Núm. 24, pp. 4802-4811
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The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination
Journal of Experimental Medicine, Vol. 209, Núm. 4, pp. 671-678
2011
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Intraclonal complexity in chronic lymphocytic leukemia: Fractions enriched in recently born/divided and older/quiescent cells
Molecular Medicine, Vol. 17, Núm. 11, pp. 1374-1382
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Mismatch-mediated error prone repair at the immunoglobulin genes
Biomedicine and Pharmacotherapy, Vol. 65, Núm. 8, pp. 529-536
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The multidimensional nature of epigenetic information and its role in disease.
Discovery medicine, Vol. 11, Núm. 58, pp. 233-243
2010
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Crosstalk between genetic and epigenetic information through cytosine deamination
Trends in Genetics, Vol. 26, Núm. 10, pp. 443-448
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MSH2/MSH6 complex promotes error-free repair of AID-Induced dU:G mispairs as well as error-prone hypermutation of A:T Sites
PLoS ONE, Vol. 5, Núm. 6
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PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance
Proceedings of the National Academy of Sciences of the United States of America, Vol. 107, Núm. 30, pp. 13384-13389
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The RNF8/RNF168 ubiquitin ligase cascade facilitates class switch recombination
Proceedings of the National Academy of Sciences of the United States of America, Vol. 107, Núm. 2, pp. 809-814
2009
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SHMTool: A webserver for comparative analysis of somatic hypermutation datasets
DNA Repair, Vol. 8, Núm. 1, pp. 137-141
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V-region mutation in vitro, in vivo, and in silico reveal the importance of the enzymatic properties of AID and the sequence environment
Proceedings of the National Academy of Sciences of the United States of America, Vol. 106, Núm. 21, pp. 8629-8634
2008
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Does antisense make sense of AID targeting?
Proceedings of the National Academy of Sciences of the United States of America
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Molecular analysis of activation-induced cytidine deaminase gene in immunoglobulin-E deficient patients
Clinical and Developmental Immunology, Vol. 2008