Publikationen, an denen er mitarbeitet A. Martínez (12)

2006

  1. Adrenomedullin is a cross-talk molecule that regulates tumor and mast cell function during human carcinogenesis

    American Journal of Pathology, Vol. 168, Núm. 1, pp. 280-291

2004

  1. Expression of complement factor H by lung cancer cells: Effects on the activation of the alternative pathway of complement

    Cancer Research, Vol. 64, Núm. 17, pp. 6310-6318

2003

  1. Mapping of the adrenomedullin-binding domains in human complement factor H

    Hypertension Research, Vol. 26, Núm. SUPPL.

2002

  1. Adrenomedullin functions as an important tumor survival factor in human carcinogenesis

    Microscopy Research and Technique, Vol. 57, Núm. 2, pp. 110-119

  2. Identification, characterization, and physiological actions of factor H as an adrenomedullin binding protein present in human plasma

    Microscopy Research and Technique, Vol. 57, Núm. 1, pp. 23-27

  3. The effects of adrenomedullin overexpression in breast tumor cells

    Journal of the National Cancer Institute, Vol. 94, Núm. 16, pp. 1226-1237

2001

  1. Cancer and diabetes: Two pathological conditions in which adrenomedullin may be involved

    Peptides, Vol. 22, Núm. 11, pp. 1719-1729

  2. Complement Factor H Is a Serum-binding Protein for Adrenomedullin, and the Resulting Complex Modulates the Bioactivities of Both Partners

    Journal of Biological Chemistry, Vol. 276, Núm. 15, pp. 12292-12300

  3. Expression of the adrenomedullin binding protein, complement factor H, in the pancreas and its physiological impact on insulin secretion

    Journal of Endocrinology, Vol. 170, Núm. 3, pp. 503-511

2000

  1. Hypoxia-inducible factor-1 (HIF-1) up-regulates adrenomedullin expression in human tumor cell lines during oxygen deprivation: A possible promotion mechanism of carcinogenesis

    Molecular Endocrinology, Vol. 14, Núm. 6, pp. 848-862

1999

  1. Is adrenomedullin a causal agent in some cases of type 2 diabetes?

    Peptides, Vol. 20, Núm. 12, pp. 1471-1478

  2. Underlying disease stress augments plasma and tissue adrenomedullin (AM) responses to endotoxin: Colocalized increases in AM and inducible nitric oxide synthase within pancreatic islets

    Endocrinology, Vol. 140, Núm. 11, pp. 5402-5411