Jesús María
Prieto Valtueña
Investigador hasta 2014
Puri
Fortes
Profesional Investigadora
Publicaciones en las que colabora con Puri Fortes (12)
2019
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The cirrhotic liver is depleted of docosahexaenoic acid (DHA), a key modulator of NF-κB and TGFβ pathways in hepatic stellate cells
Cell Death and Disease, Vol. 10, Núm. 1
2018
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TMEM173 Alternative Spliced Isoforms Modulate Viral Replication through the STING Pathway
ImmunoHorizons, Vol. 2, Núm. 11, pp. 363-376
2016
2012
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AAV vectors transduce hepatocytes in vivo as efficiently in cirrhotic as in healthy rat livers
Gene Therapy, Vol. 19, Núm. 4, pp. 411-417
2010
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Insulin-like growth factor i gene transfer to cirrhotic liver induces fibrolysis and reduces fibrogenesis leading to cirrhosis reversion in rats
Hepatology, Vol. 51, Núm. 3, pp. 912-921
2006
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Cardiotrophin-1 defends the liver against ischemia-reperfusion injury and mediates the protective effect of ischemic preconditioning
Journal of Experimental Medicine, Vol. 203, Núm. 13, pp. 2809-2815
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Effect of adenovirus-mediated RNA interference on endogenous microRNAs in a mouse model of multidrug resistance protein 2 gene silencing
Journal of Virology, Vol. 80, Núm. 24, pp. 12236-12247
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Low surface expression of B7-1 (CD80) is an immunoescape mechanism of colon carcinoma
Cancer Research, Vol. 66, Núm. 4, pp. 2442-2450
2005
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Intratumoral injection of dendritic cells transduced by an SV40-based vector expressing interleukin-15 induces curative immunity mediated by CD8+ T lymphocytes and NK cells
Molecular Therapy, Vol. 12, Núm. 5, pp. 950-959
2004
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Factors influencing the production of recombinant SV40 vectors
Molecular Therapy, Vol. 10, Núm. 4, pp. 780-791
2003
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In vitro and in vivo comparative study of chimeric liver-specific promoters
Molecular Therapy, Vol. 7, Núm. 3, pp. 375-385
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Inhibiting expression of specific genes in mammalian cells with 5′ end-mutated U1 small nuclear RNAs targeted to terminal exons of pre-MRNA
Proceedings of the National Academy of Sciences of the United States of America, Vol. 100, Núm. 14, pp. 8264-8269