Publications (35) Maite Mendívil Pérez publications

2024

  1. APOE3 Christchurch modulates β-catenin/Wnt signaling in iPS cell-derived cerebral organoids from Alzheimer’s cases

    Frontiers in Molecular Neuroscience, Vol. 17

  2. Neuroprotective Effect of Combined Treatment with Epigallocatechin 3-Gallate and Melatonin on Familial Alzheimer’s Disease PSEN1 E280A Cerebral Spheroids Derived from Menstrual Mesenchymal Stromal Cells

    Journal of Alzheimer's Disease, Vol. 99, Núm. s1, pp. S51-S66

  3. Rotenone Induces a Neuropathological Phenotype in Cholinergic-like Neurons Resembling Parkinson’s Disease Dementia (PDD)

    Neurotoxicity Research, Vol. 42, Núm. 3

  4. The GBA1 K198E Variant Is Associated with Suppression of Glucocerebrosidase Activity, Autophagy Impairment, Oxidative Stress, Mitochondrial Damage, and Apoptosis in Skin Fibroblasts

    International Journal of Molecular Sciences, Vol. 25, Núm. 17

  5. Toxicology of anhydroecgonine methyl ester: A systematic review of a cocaine pyrolysis product

    Toxicology Reports, Vol. 13

2023

  1. Cannabidiol Protects Dopaminergic-like Neurons against Paraquat- and Maneb-Induced Cell Death through Safeguarding DJ-1CYS106 and Caspase 3 Independently of Cannabinoid Receptors: Relevance in Parkinson’s Disease

    ACS Chemical Neuroscience, Vol. 14, Núm. 11, pp. 2159-2171

  2. Cholinergic-like neurons and cerebral spheroids bearing the PSEN1 p.Ile416Thr variant mirror Alzheimer's disease neuropathology

    Scientific Reports, Vol. 13, Núm. 1

  3. Levels of burnout and exposure to ethical conflict and assessment of the practice environment in nursing professionals of intensive care

    Enfermeria Intensiva, Vol. 34, Núm. 4, pp. 195-204

  4. PSEN1 E280A Cholinergic-like Neurons and Cerebral Spheroids Derived from Mesenchymal Stromal Cells and from Induced Pluripotent Stem Cells Are Neuropathologically Equivalent

    International Journal of Molecular Sciences, Vol. 24, Núm. 10

  5. Rotenone Blocks the Glucocerebrosidase Enzyme and Induces the Accumulation of Lysosomes and Autophagolysosomes Independently of LRRK2 Kinase in HEK-293 Cells

    International Journal of Molecular Sciences, Vol. 24, Núm. 13

  6. TPEN/TPGS (T2) combo dramatically reduces Philadelphia chromosome-positive pro-lymphoblastic B leukemia in BALB/c mice

    Medical Oncology, Vol. 40, Núm. 1

2022

  1. Combinational treatment of TPEN and TPGS induces apoptosis in acute lymphoblastic and chronic myeloid leukemia cells in vitro and ex vivo

    Medical Oncology, Vol. 39, Núm. 7

  2. Polycationic peptide R7-G-Aβ25-35 selectively induces cell death in leukemia Jurkat T cells through speedy mitochondrial depolarization, and CASPASE-3 -independent mechanism

    Biochemistry and Biophysics Reports, Vol. 31

  3. TPEN selectively eliminates lymphoblastic B cells from bone marrow pediatric acute lymphoblastic leukemia patients

    BioMetals, Vol. 35, Núm. 4, pp. 741-758

2021

  1. (−)-epigallocatechin-3-gallate diminishes intra-and extracellular amyloid-induced cytotoxic effects on cholinergic-like neurons from familial Alzheimer’s disease PSEN1 E280A

    Biomolecules, Vol. 11, Núm. 12

  2. Latent Tri-lineage Potential of Human Menstrual Blood–Derived Mesenchymal Stromal Cells Revealed by Specific In Vitro Culture Conditions

    Molecular Neurobiology, Vol. 58, Núm. 10, pp. 5194-5209

  3. Multi-Target Effects of the Cannabinoid CP55940 on Familial Alzheimer's Disease PSEN1 E280A Cholinergic-Like Neurons: Role of CB1 Receptor

    Journal of Alzheimer's Disease, Vol. 82, Núm. s1, pp. S359-S378

  4. TPEN exerts selective anti-leukemic efficacy in ex vivo drug-resistant childhood acute leukemia

    BioMetals, Vol. 34, Núm. 1, pp. 49-66

2020

  1. Cannabinoid CP55940 selectively induces apoptosis in Jurkat cells and in ex vivo T-cell acute lymphoblastic leukemia through H2O2 signaling mechanism

    Leukemia Research, Vol. 95

  2. Cholinergic-like neurons carrying PSEN1 E280A mutation from familial Alzheimer’s disease reveal intraneuronal sAPPβ fragments accumulation, hyperphosphorylation of TAU, oxidative stress, apoptosis and Ca2+ dysregulation: Therapeutic implications

    PLoS ONE, Vol. 15, Núm. 5