Publicaciones en colaboración con investigadores/as de Icahn School of Medicine at Mount Sinai (7)

2018

  1. High Mobility Group Box-1 Drives Fibrosis Progression Signaling via the Receptor for Advanced Glycation End Products in Mice

    Hepatology, Vol. 68, Núm. 6, pp. 2380-2404

2017

  1. Signalling via the osteopontin and high mobility group box-1 axis drives the fibrogenic response to liver injury

    Gut, Vol. 66, Núm. 6, pp. 1123-1137

2016

  1. Cartilage oligomeric matrix protein participates in the pathogenesis of liver fibrosis

    Journal of Hepatology, Vol. 65, Núm. 5, pp. 963-971

2014

  1. Extracellular matrix and liver disease

    Antioxidants and Redox Signaling, Vol. 21, Núm. 7, pp. 1078-1097

  2. High Mobility Group Box-1 (HMGB1) participates in the pathogenesis of Alcoholic Liver Disease (ALD)

    Journal of Biological Chemistry, Vol. 289, Núm. 33, pp. 22672-22691

  3. Osteopontin binding to lipopolysaccharide lowers tumor necrosis factor-α and prevents early alcohol-induced liver injury in mice

    Hepatology, Vol. 59, Núm. 4, pp. 1600-1616

2013

  1. GCN2 kinase is a key regulator of fibrogenesis and acute and chronic liver injury induced by carbon tetrachloride in mice

    Laboratory Investigation, Vol. 93, Núm. 3, pp. 303-310