María Cruz
Rodríguez Oroz
Consultora Médica
Institut d'Investigacions Biomèdiques August Pi i Sunyer
Barcelona, EspañaPublicacións en colaboración con investigadores/as de Institut d'Investigacions Biomèdiques August Pi i Sunyer (12)
2020
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Correction to: Motor impulsivity and delay intolerance are elicited in a dose-dependent manner with a dopaminergic agonist in parkinsonian rats (Psychopharmacology, (2020), 237, 8, (2419-2431), 10.1007/s00213-020-05544-6)
Psychopharmacology
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Motor impulsivity and delay intolerance are elicited in a dose-dependent manner with a dopaminergic agonist in parkinsonian rats
Psychopharmacology, Vol. 237, Núm. 8, pp. 2419-2431
2019
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Pramipexole-induced impulsivity in mildparkinsonian rats: a model of impulse control disorders in Parkinson's disease
Neurobiology of Aging, Vol. 75, pp. 126-135
2015
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Dyskinesias and impulse control disorders in Parkinson's disease: From pathogenesis to potential therapeutic approaches
Neuroscience and Biobehavioral Reviews, Vol. 56, pp. 294-314
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From unilateral to bilateral parkinsonism: Effects of lateralization on dyskinesias and associated molecular mechanisms
Neuropharmacology, Vol. 97, pp. 365-375
2013
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Subthalamic 6-OHDA-induced lesion attenuates levodopa-induced dyskinesias in the rat model of Parkinson's disease
Experimental Neurology, Vol. 250, pp. 304-312
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l-dopa-induced dyskinesias in unilateral 6-hydroxydopamine-lesioned rats are not modified by excitotoxic lesion of the entopeduncular nucleus and substantia nigra pars reticulata
Synapse, Vol. 67, Núm. 7, pp. 407-414
2011
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Continuous dopaminergic stimulation: Clinical aspects and experimental bases
Neurologist, Vol. 17, Núm. 6 SUPPL.
2009
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Local administration of sarizotan into the subthalamic nucleus attenuates levodopa-induced dyskinesias in 6-OHDA-lesioned rats
Psychopharmacology, Vol. 204, Núm. 2, pp. 241-250
2008
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Entacapone potentiates the long-duration response but does not normalize levodopa-induced molecular changes
Neurobiology of Disease, Vol. 32, Núm. 3, pp. 340-348
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Functional organization of the basal ganglia: Therapeutic implications for Parkinson's disease
Movement Disorders, Vol. 23, Núm. SUPPL. 3