María José
Calasanz Abinzano
Catedrática de Universidad
Felipe
Prosper Cardoso
Consultor Médico
Publicaciones en las que colabora con Felipe Prosper Cardoso (66)
2024
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Epigenetic-based differentiation therapy for Acute Myeloid Leukemia
Nature Communications, Vol. 15, Núm. 1
2023
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Integrated flow cytometry and sequencing to reconstruct evolutionary patterns from dysplasia to acute myeloid leukemia
Blood Advances, Vol. 7, Núm. 1, pp. 167-173
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Preclinical models for prediction of immunotherapy outcomes and immune evasion mechanisms in genetically heterogeneous multiple myeloma
Nature Medicine, Vol. 29, Núm. 3, pp. 632-645
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Transcriptional and genomic characterization of measurable residual disease in acute myeloid leukaemia
British Journal of Haematology
2022
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Insights into the mechanisms underlying aberrant SOX11 oncogene expression in mantle cell lymphoma
Leukemia, Vol. 36, Núm. 2, pp. 583-587
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Landscape and clinical significance of long noncoding RNAs involved in multiple myeloma expressed fusion transcripts
American Journal of Hematology
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Preneoplastic somatic mutations including MYD88L265P in lymphoplasmacytic lymphoma
Science Advances, Vol. 8, Núm. 3
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The transcriptomic landscape of elderly acute myeloid leukemia identifies B7H3 and BANP as a favorable signature in high-risk patients
Frontiers in Oncology, Vol. 12
2021
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Characterization of complete lncRNAs transcriptome reveals the functional and clinical impact of lncRNAs in multiple myeloma
Leukemia, Vol. 35, Núm. 5, pp. 1438-1450
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Endogenous retroelement activation by epigenetic therapy reverses the warburg effect and elicits mitochondrial-mediated cancer cell death
Cancer Discovery, Vol. 11, Núm. 5, pp. 1268-1285
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Measurable residual disease in elderly acute myeloid leukemia: Results from the PETHEMA-FLUGAZA phase 3 clinical trial
Blood Advances, Vol. 5, Núm. 3, pp. 760-770
2020
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Assessment of minimal residual disease by next generation sequencing in peripheral blood as a complementary tool for personalized transplant monitoring in myeloid neoplasms
Journal of Clinical Medicine, Vol. 9, Núm. 12, pp. 1-20
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Assessment of the clinical utility of four NGS panels in myeloid malignancies. Suggestions for NGS panel choice or design
PLoS ONE, Vol. 15, Núm. 1
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Chromatin activation as a unifying principle underlying pathogenic mechanisms in multiple myeloma
Genome Research, Vol. 30, Núm. 9, pp. 1217-1227
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Circulating tumor cells for comprehensive and multiregional non-invasive genetic characterization of multiple myeloma
Leukemia, Vol. 34, Núm. 11, pp. 3007-3018
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Strategy for identification of a potential inherited leukemia predisposition in a 299 patient's cohort with tumor-only sequencing data
Leukemia Research, Vol. 95
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Transcriptional profiling of circulating tumor cells in multiple myeloma: a new model to understand disease dissemination
Leukemia, Vol. 34, Núm. 2, pp. 589-603
2019
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Epigenomic profiling of myelofibrosis reveals widespread DNA methylation changes in enhancer elements and ZFP36L1 as a potential tumor suppressor gene that is epigenetically regulated
Haematologica, Vol. 104, Núm. 8, pp. 1572-1579
2018
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Richter transformation driven by Epstein–Barr virus reactivation during therapy-related immunosuppression in chronic lymphocytic leukaemia
Journal of Pathology, Vol. 245, Núm. 1, pp. 61-73