Centro de Investigación Biomédica en Red sobre Enfermedades Hepáticas y Digestivas-ko ikertzaileekin lankidetzan egindako argitalpenak (7)

2018

  1. Erratum to “A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction” [J Hepatol 67 (2017) 669–679] (S0168827817320469) (10.1016/j.jhep.2017.05.010))

    Journal of Hepatology

2017

  1. A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction

    Journal of Hepatology, Vol. 67, Núm. 4, pp. 669-679

2012

  1. Matrigel-embedded 3D culture of Huh-7 cells as a hepatocyte-like polarized system to study hepatitis C virus cycle

    Virology, Vol. 425, Núm. 1, pp. 31-39

  2. N-terminal acetylome analyses and functional insights of the N-terminal acetyltransferase NatB

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 109, Núm. 31, pp. 12449-12454

2011

  1. Hepatitis C virus induces the expression of CCL17 and CCL22 chemokines that attract regulatory T cells to the site of infection

    Journal of Hepatology, Vol. 54, Núm. 3, pp. 422-431

2009

  1. The tight junction-associated protein occludin is required for a postbinding step in hepatitis C virus entry and infection

    Journal of Virology, Vol. 83, Núm. 16, pp. 8012-8020

2008

  1. Hepatitis C virus envelope components alter localization of hepatocyte tight junction-Associated proteins and promote occludin retention in the endoplasmic reticulum

    Hepatology, Vol. 48, Núm. 4, pp. 1044-1053