Publicaciones en las que colabora con Víctor Valentí Azcarate (71)

2024

  1. Decreased expression of the NLRP6 inflammasome is associated with increased intestinal permeability and inflammation in obesity with type 2 diabetes

    Cellular and Molecular Life Sciences, Vol. 81, Núm. 1

  2. Differential mitochondrial adaptation and FNDC5 production in brown and white adipose tissue in response to cold and obesity

    Obesity

  3. FNDC4 reduces hepatocyte inflammatory cell death via AMPKα in metabolic dysfunction-associated steatotic liver disease

    Clinical Nutrition, Vol. 43, Núm. 9, pp. 2221-2233

  4. Single anastomosis duodeno-ileal bypass with sleeve gastrectomy generates sustained improvement of glycemic control compared with sleeve gastrectomy in the diet-induced obese rat model

    Journal of Physiology and Biochemistry, Vol. 80, Núm. 1, pp. 149-160

2023

  1. Antagonic effect of ghrelin and LEAP-2 on hepatic stellate cell activation and liver fibrosis in obesity-associated nonalcoholic fatty liver disease

    European journal of endocrinology, Vol. 188, Núm. 7, pp. 564-577

  2. Corrigendum: Increased levels of interleukin-36 in obesity and type 2 diabetes fuels adipose tissue inflammation by inducing its own expression and release by adipocytes and macrophages(Front. Immunol., (2022), 13, (832185), 10.3389/fimmu.2022.832185)

    Frontiers in Immunology

  3. Effect of guanylin peptides on pancreas steatosis and function in experimental diet-induced obesity and after bariatric surgery

    Frontiers in Endocrinology, Vol. 14

  4. Increased Aquaporin-7 Expression Is Associated with Changes in Rat Brown Adipose Tissue Whitening in Obesity: Impact of Cold Exposure and Bariatric Surgery

    International Journal of Molecular Sciences, Vol. 24, Núm. 4

  5. Increased Expression Levels of Netrin-1 in Visceral Adipose Tissue during Obesity Favour Colon Cancer Cell Migration

    Cancers, Vol. 15, Núm. 4

  6. Uroguanylin prevents hepatic steatosis, mitochondrial dysfunction and fibrosis in obesity-associated NAFLD

    Metabolism: Clinical and Experimental, Vol. 147

2022

  1. Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements

    Acta Biomaterialia, Vol. 141, pp. 264-279

  2. Dermatopontin Influences the Development of Obesity-Associated Colon Cancer by Changes in the Expression of Extracellular Matrix Proteins

    International Journal of Molecular Sciences, Vol. 23, Núm. 16

  3. Improved Adipose Tissue Function after Single Anastomosis Duodeno-Ileal Bypass with Sleeve-Gastrectomy (SADI-S) in Diet-Induced Obesity

    International Journal of Molecular Sciences, Vol. 23, Núm. 19

  4. Increased Levels of Interleukin-36 in Obesity and Type 2 Diabetes Fuel Adipose Tissue Inflammation by Inducing Its Own Expression and Release by Adipocytes and Macrophages

    Frontiers in Immunology, Vol. 13

  5. Netrin-1 Promotes Visceral Adipose Tissue Inflammation in Obesity and Is Associated with Insulin Resistance

    Nutrients, Vol. 14, Núm. 20

  6. Serum Levels of IL-1 RA Increase with Obesity and Type 2 Diabetes in Relation to Adipose Tissue Dysfunction and are Reduced After Bariatric Surgery in Parallel to Adiposity

    Journal of Inflammation Research, Vol. 15, pp. 1331-1345

2021

  1. Decreased levels of microfibril‐associated glycoprotein (Magp)‐1 in patients with colon cancer and obesity are associated with changes in extracellular matrix remodelling

    International Journal of Molecular Sciences, Vol. 22, Núm. 16

  2. FNDC4 and FNDC5 reduce SARS-CoV-2 entry points and spike glycoprotein S1-induced pyroptosis, apoptosis, and necroptosis in human adipocytes

    Cellular and Molecular Immunology, Vol. 18, Núm. 10, pp. 2457-2459

  3. NLRP3 inflammasome blockade reduces adipose tissue inflammation and extracellular matrix remodeling

    Cellular and Molecular Immunology, Vol. 18, Núm. 4, pp. 1045-1057

  4. Obese patients with NASH have increased hepatic expression of SARS-CoV-2 critical entry points

    Journal of Hepatology