Publications en collaboration avec des chercheurs de Mayo Clinic (22)

2024

  1. KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis

    Cancer Research Communications, Vol. 4, Núm. 7, pp. 1677-1689

2020

  1. GLI1/GLI2 functional interplay is required to control Hedgehog/GLI targets gene expression

    Biochemical Journal, Vol. 477, Núm. 17, pp. 3131-3145

  2. Proteostasis disturbances and endoplasmic reticulum stress contribute to polycystic liver disease: New therapeutic targets

    Liver International, Vol. 40, Núm. 7, pp. 1670-1685

2018

  1. MicroRNA-506 promotes primary biliary cholangitis–like features in cholangiocytes and immune activation

    Hepatology, Vol. 67, Núm. 4, pp. 1420-1440

2017

  1. IKBKE is required during KRAS-induced pancreatic tumorigenesis

    Cancer Research, Vol. 77, Núm. 2, pp. 320-329

  2. SOX17 regulates cholangiocyte differentiation and acts as a tumor suppressor in cholangiocarcinoma

    Journal of Hepatology, Vol. 67, Núm. 1, pp. 72-83

2016

  1. Nuclear factor of activated T cells-dependent downregulation of the transcription factor glioma-associated protein 1 (GLI1) underlies the growth inhibitory properties of arachidonic acid

    Journal of Biological Chemistry, Vol. 291, Núm. 4, pp. 1933-1947

  2. Regulation of GLI underlies a role for BET bromodomains in pancreatic cancer growth and the tumor microenvironment

    Clinical Cancer Research, Vol. 22, Núm. 16, pp. 4259-4270

2014

  1. Discovering and targeting the epigenetic pathways to treat muscle loss: Working toward a paradigm shift in cancer therapeutics

    Current Opinion in Supportive and Palliative Care

  2. Galectin-1 drives pancreatic carcinogenesis through stroma remodeling and hedgehog signaling activation

    Cancer Research, Vol. 74, Núm. 13, pp. 3512-3524

  3. Identification of novel non-coding RNA-based negative feedback regulating the expression of the oncogenic transcription factor GLI1

    Molecular Oncology, Vol. 8, Núm. 5, pp. 912-926

  4. Inactivation of the transcription factor GLI1 accelerates pancreatic cancer progression

    Journal of Biological Chemistry, Vol. 289, Núm. 23, pp. 16516-16525

  5. Inhibition of metalloprotease hyperactivity in cystic cholangiocytes halts the development of polycystic liver diseases

    Gut, Vol. 63, Núm. 10, pp. 1658-1667

  6. Stromal elements act to restrain, rather than support, pancreatic ductal adenocarcinoma

    Cancer Cell, Vol. 25, Núm. 6, pp. 735-747

  7. The transcription factor GLI1 interacts with SMAD proteins to modulate transforming growth factor β-induced gene expression in a p300/CREB-binding protein-associated factor (PCAF)-dependent manner

    Journal of Biological Chemistry, Vol. 289, Núm. 22, pp. 15495-15506

  8. The transcription factor GLI1 modulates the inflammatory response during pancreatic tissue remodeling

    Journal of Biological Chemistry, Vol. 289, Núm. 40, pp. 27727-27743

2013

  1. Activation of the transcription factor gli1 by wnt signaling underlies the role of SULFATASE 2 as a regulator of tissue regeneration

    Journal of Biological Chemistry, Vol. 288, Núm. 29, pp. 21389-21398

2012

  1. GLI1 inhibition promotes epithelial-to-mesenchymal transition in pancreatic cancer cells

    Cancer Research, Vol. 72, Núm. 1, pp. 88-99

  2. GLI1 modulates EMT in pancreatic cancer - Response

    Cancer Research

  3. MicroRNAs in biliary diseases

    World Journal of Gastroenterology, Vol. 18, Núm. 43, pp. 6189-6196