Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas-ko ikertzaileekin lankidetzan egindako argitalpenak (8)

2022

  1. Genetic Inactivation of Free Fatty Acid Receptor 3 Impedes Behavioral Deficits and Pathological Hallmarks in the APPswe Alzheimer’s Disease Mouse Model

    International Journal of Molecular Sciences, Vol. 23, Núm. 7

2020

  1. Reversal of Object Recognition Memory Deficit in Perirhinal Cortex-Lesioned Rats and Primates and in Rodent Models of Aging and Alzheimer's Diseases

    Neuroscience, Vol. 448, pp. 287-298

  2. Smelling the Dark Proteome: Functional Characterization of PITH Domain-Containing Protein 1 (C1orf128) in Olfactory Metabolism

    Journal of Proteome Research, Vol. 19, Núm. 12, pp. 4826-4843

2018

  1. Maternal imprinting on cognition markers of wild type and transgenic Alzheimer's disease model mice

    Scientific Reports, Vol. 8, Núm. 1

2016

  1. Limited unfolded protein response and inflammation in neuroserpinopathy

    Journal of Neuropathology and Experimental Neurology, Vol. 75, Núm. 2, pp. 121-133

2011

  1. Enhanced expression of the voltage-dependent anion channel 1 (VDAC1) in alzheimer's disease transgenic mice: An insight into the pathogenic effects of amyloid-β

    Journal of Alzheimer's Disease, Vol. 23, Núm. 2, pp. 195-206

2009

  1. Early changes in hippocampal eph receptors precede the onset of memory decline in mouse models of alzheimer's disease

    Journal of Alzheimer's Disease, Vol. 17, Núm. 4, pp. 773-786

  2. Overexpression of wild-type human APP in mice causes cognitive deficits and pathological features unrelated to Aβ levels

    Neurobiology of Disease, Vol. 33, Núm. 3, pp. 369-378