Publikationen (40) Publikationen von Alberto Pérez Mediavilla

2023

  1. Stabilization of 14-3-3 protein-protein interactions with Fusicoccin-A decreases alpha-synuclein dependent cell-autonomous death in neuronal and mouse models

    Neurobiology of Disease, Vol. 183

  2. Síntesis y modificación de proteínas

    Biología celular biomédica (Elsevier), pp. 198-221

2022

  1. Genetic Inactivation of Free Fatty Acid Receptor 3 Impedes Behavioral Deficits and Pathological Hallmarks in the APPswe Alzheimer’s Disease Mouse Model

    International Journal of Molecular Sciences, Vol. 23, Núm. 7

  2. The Cognitive Improvement and Alleviation of Brain Hypermetabolism Caused by FFAR3 Ablation in Tg2576 Mice Is Persistent under Diet-Induced Obesity

    International Journal of Molecular Sciences, Vol. 23, Núm. 21

2021

  1. Identifying the main functional pathways associated with cognitive resilience to alzheimer’s disease

    International Journal of Molecular Sciences, Vol. 22, Núm. 17

  2. Maternal imprinting, mitochondrial DNA, nuclear DNA and Alzheimer’s disease

    Exploration of Neuroprotective Therapy, Vol. 1, Núm. 2, pp. 121-126

2020

  1. Reversal of Object Recognition Memory Deficit in Perirhinal Cortex-Lesioned Rats and Primates and in Rodent Models of Aging and Alzheimer's Diseases

    Neuroscience, Vol. 448, pp. 287-298

  2. Smelling the Dark Proteome: Functional Characterization of PITH Domain-Containing Protein 1 (C1orf128) in Olfactory Metabolism

    Journal of Proteome Research, Vol. 19, Núm. 12, pp. 4826-4843

2019

  1. Bases moleculares de las enfermedades neurodegenerativas

    Principios de bioquímica clínica y patología molecular (Elsevier España), pp. 391-404

  2. Early-onset molecular derangements in the olfactory bulb of Tg2576 mice: Novel insights into the stress-responsive olfactory kinase dynamics in Alzheimer's disease

    Frontiers in Aging Neuroscience, Vol. 11, Núm. JUN

2018

  1. Maternal imprinting on cognition markers of wild type and transgenic Alzheimer's disease model mice

    Scientific Reports, Vol. 8, Núm. 1

2017

  1. Network-driven proteogenomics unveils an aging-related imbalance in the olfactory IκBα-NFκB p65 complex functionality in Tg2576 alzheimer’s disease mouse model

    International Journal of Molecular Sciences, Vol. 18, Núm. 11

2016

  1. Limited unfolded protein response and inflammation in neuroserpinopathy

    Journal of Neuropathology and Experimental Neurology, Vol. 75, Núm. 2, pp. 121-133

2014

  1. Expression of the Glucose Transporter GLUT12 in Alzheimer's Disease Patients

    Journal of Alzheimer's Disease, Vol. 42, Núm. 1, pp. 97-101

  2. GPR40 activation leads to CREB and ERK phosphorylation in primary cultures of neurons from the mouse CNS and in human neuroblastoma cells

    Hippocampus, Vol. 24, Núm. 7, pp. 733-739

2013

  1. Age-related mitochondrial alterations without neuronal loss in the hippocampus of a transgenic model of Alzheimer's disease

    Current Alzheimer Research, Vol. 10, Núm. 4, pp. 390-405

  2. Phenyl Acyl Acids Attenuate the Unfolded Protein Response in Tunicamycin-Treated Neuroblastoma Cells

    PLoS ONE, Vol. 8, Núm. 8

2012

  1. Accelerated aging of the GABAergic septohippocampal pathway and decreased hippocampal rhythms in a mouse model of Alzheimer's disease

    FASEB Journal, Vol. 26, Núm. 11, pp. 4458-4467

  2. Chronic mild stress accelerates the onset and progression of the Alzheimer's disease phenotype in Tg2576 mice

    Journal of Alzheimer's Disease, Vol. 28, Núm. 3, pp. 567-578

2011

  1. Chronic mild stress in mice promotes cognitive impairment and CDK5-dependent tau hyperphosphorylation

    Behavioural Brain Research, Vol. 220, Núm. 2, pp. 338-343