Publicaciones en las que colabora con Víctor Valentí Azcarate (52)

2024

  1. Decreased expression of the NLRP6 inflammasome is associated with increased intestinal permeability and inflammation in obesity with type 2 diabetes

    Cellular and Molecular Life Sciences, Vol. 81, Núm. 1

  2. FNDC4 reduces hepatocyte inflammatory cell death via AMPKα in metabolic dysfunction-associated steatotic liver disease

    Clinical Nutrition, Vol. 43, Núm. 9, pp. 2221-2233

2023

  1. Antagonic effect of ghrelin and LEAP-2 on hepatic stellate cell activation and liver fibrosis in obesity-associated nonalcoholic fatty liver disease

    European journal of endocrinology, Vol. 188, Núm. 7, pp. 564-577

  2. Corrigendum: Increased levels of interleukin-36 in obesity and type 2 diabetes fuels adipose tissue inflammation by inducing its own expression and release by adipocytes and macrophages(Front. Immunol., (2022), 13, (832185), 10.3389/fimmu.2022.832185)

    Frontiers in Immunology

  3. Downregulated Adipose Tissue Expression of Browning Genes With Increased Environmental Temperatures

    The Journal of clinical endocrinology and metabolism, Vol. 109, Núm. 1, pp. e145-e154

  4. Uroguanylin prevents hepatic steatosis, mitochondrial dysfunction and fibrosis in obesity-associated NAFLD

    Metabolism: Clinical and Experimental, Vol. 147

2022

  1. Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements

    Acta Biomaterialia, Vol. 141, pp. 264-279

  2. Dermatopontin Influences the Development of Obesity-Associated Colon Cancer by Changes in the Expression of Extracellular Matrix Proteins

    International Journal of Molecular Sciences, Vol. 23, Núm. 16

  3. Netrin-1 Promotes Visceral Adipose Tissue Inflammation in Obesity and Is Associated with Insulin Resistance

    Nutrients, Vol. 14, Núm. 20

2021

  1. Decreased levels of microfibril‐associated glycoprotein (Magp)‐1 in patients with colon cancer and obesity are associated with changes in extracellular matrix remodelling

    International Journal of Molecular Sciences, Vol. 22, Núm. 16

  2. FNDC4 and FNDC5 reduce SARS-CoV-2 entry points and spike glycoprotein S1-induced pyroptosis, apoptosis, and necroptosis in human adipocytes

    Cellular and Molecular Immunology, Vol. 18, Núm. 10, pp. 2457-2459

  3. NLRP3 inflammasome blockade reduces adipose tissue inflammation and extracellular matrix remodeling

    Cellular and Molecular Immunology, Vol. 18, Núm. 4, pp. 1045-1057

  4. Role of ANGPTL8 in NAFLD improvement after bariatric surgery in experimental and human obesity

    International Journal of Molecular Sciences, Vol. 22, Núm. 23

  5. The differential expression of the inflammasomes in adipose tissue and colon influences the development of colon cancer in a context of obesity by regulating intestinal inflammation

    Journal of Inflammation Research, Vol. 14, pp. 6431-6446

2020

  1. Aquaporin-11 Contributes to TGF-β1-Induced Endoplasmic Reticulum Stress in Human Visceral Adipocytes: Role in Obesity-Associated Inflammation

    Cells, Vol. 9, Núm. 6

  2. Dermatopontin, a novel adipokine promoting adipose tissue extracellular matrix remodelling and inflammation in obesity

    Journal of Clinical Medicine, Vol. 9, Núm. 4

  3. FNDC4, a novel adipokine that reduces lipogenesis and promotes fat browning in human visceral adipocytes

    Metabolism: Clinical and Experimental, Vol. 108

  4. Ghrelin Reduces TNF-α-Induced Human Hepatocyte Apoptosis, Autophagy, and Pyroptosis: Role in Obesity-Associated NAFLD

    Journal of Clinical Endocrinology and Metabolism, Vol. 104, Núm. 1, pp. 21-37

  5. Impact of Routine and Long-Term Follow-Up on Weight Loss after Bariatric Surgery

    Obesity Surgery, Vol. 30, Núm. 11, pp. 4293-4299

2019

  1. GLP-1 limits adipocyte inflammation and its low circulating pre-operative concentrations predict worse type 2 diabetes remission after bariatric surgery in obese patients

    Journal of Clinical Medicine, Vol. 8, Núm. 4