(FM) Medicina Interna
Departamento académico
Tomás
Aragón Amonarriz
Profesional Investigador
Publicaciones en las que colabora con Tomás Aragón Amonarriz (13)
2023
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Author Correction: Defective HNF4alpha-dependent gene expression as a driver of hepatocellular failure in alcoholic hepatitis (Nature Communications, (2019), 10, 1, (3126), 10.1038/s41467-019-11004-3)
Nature Communications
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Stabilization of 14-3-3 protein-protein interactions with Fusicoccin-A decreases alpha-synuclein dependent cell-autonomous death in neuronal and mouse models
Neurobiology of Disease, Vol. 183
2021
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Linking the Expression of Therapeutic Genes to Unfolded Protein Response: A New Option for Anti-Hepatitis B Virus Gene Therapy
Human Gene Therapy, Vol. 32, Núm. 7-8, pp. 341-348
2020
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Maturation of NAA20 Aminoterminal End Is Essential to Assemble NatB N-Terminal Acetyltransferase Complex
Journal of Molecular Biology, Vol. 432, Núm. 22, pp. 5889-5901
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N-terminal acetylation mutants affect alpha-synuclein stability, protein levels and neuronal toxicity
Neurobiology of Disease, Vol. 137
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Proteostasis disturbances and endoplasmic reticulum stress contribute to polycystic liver disease: New therapeutic targets
Liver International, Vol. 40, Núm. 7, pp. 1670-1685
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TNF-alpha inhibition ameliorates HDV-induced liver damage in a mouse model of acute severe infection
JHEP Reports, Vol. 2, Núm. 3
2019
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Defective HNF4alpha-dependent gene expression as a driver of hepatocellular failure in alcoholic hepatitis
Nature Communications, Vol. 10, Núm. 1
2018
2017
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Iron affects Ire1 clustering propensity and the amplitude of endoplasmic reticulum stress signaling
Journal of Cell Science, Vol. 130, Núm. 19, pp. 3222-3233
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X-box Binding Protein 1 Regulates Unfolded Protein, Acute-Phase, and DNA Damage Responses During Regeneration of Mouse Liver
Gastroenterology, Vol. 152, Núm. 5, pp. 1203-1216.e15
2014
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Cardiotrophin-1 eliminates hepatic steatosis in obese mice by mechanisms involving AMPK activation
Journal of Hepatology, Vol. 60, Núm. 5, pp. 1017-1025
2013
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Helper-dependent adenoviral liver gene therapy protects against induced attacks and corrects protein folding stress in acute intermittent porphyria mice
Human Molecular Genetics, Vol. 22, Núm. 14, pp. 2929-2940