Publikationen, an denen er mitarbeitet Estefanía Rodríguez García (11)

2022

  1. Optimising the IgG-degrading enzyme treatment regimen for enhanced adeno-associated virus transduction in the presence of neutralising antibodies

    Clinical and Translational Immunology, Vol. 11, Núm. 2

2021

  1. AdrA as a Potential Immunomodulatory Candidate for STING-Mediated Antiviral Therapy That Required Both Type I IFN and TNF-α Production

    Journal of immunology (Baltimore, Md. : 1950), Vol. 206, Núm. 2, pp. 376-385

  2. Epitope spreading driven by the joint action of CART cells and pharmacological STING stimulation counteracts tumor escape via antigen-loss variants

    Journal for ImmunoTherapy of Cancer, Vol. 9, Núm. 11

  3. Linking the Expression of Therapeutic Genes to Unfolded Protein Response: A New Option for Anti-Hepatitis B Virus Gene Therapy

    Human Gene Therapy, Vol. 32, Núm. 7-8, pp. 341-348

2018

  1. TMEM173 Alternative Spliced Isoforms Modulate Viral Replication through the STING Pathway

    ImmunoHorizons, Vol. 2, Núm. 11, pp. 363-376

2017

  1. Successful Repeated Hepatic Gene Delivery in Mice and Non-human Primates Achieved by Sequential Administration of AAV5ch and AAV1

    Molecular Therapy, Vol. 25, Núm. 8, pp. 1831-1842

2016

  1. A versatile vector for in vivo monitoring of type I interferon induction and signaling

    PLoS ONE, Vol. 11, Núm. 3

  2. Molecular mechanisms and biological relevance of sting in antiviral innate immunity activation and immunostimulation

    Molecular mechanisms and biological relevance of sting in antiviral innate immunity activation and immunostimulation

  3. Transient expression of transgenic IL-12 in mouse liver triggers unremitting inflammation mimicking human autoimmune hepatitis

    Journal of Immunology, Vol. 197, Núm. 6, pp. 2145-2156

2015

  1. A RIG-I 2CARD-MAVS200 Chimeric Protein Reconstitutes IFN-β Induction and Antiviral Response in Models Deficient in Type i IFN Response

    Journal of Innate Immunity, Vol. 7, pp. 466-481

2014

  1. Conventional but not plasmacytoid dendritic cells foster the systemic virus-induced type I IFN response needed for efficient CD8 T cell priming

    Journal of Immunology, Vol. 193, Núm. 3, pp. 1151-1161