(FM) Medicina Interna
Departamento académico
Francisco Javier
Cubero Palero
Publikationen, an denen er mitarbeitet Francisco Javier Cubero Palero (17)
2024
2023
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Advancing with cancer immunotherapeutics: CD29+ regulatory T cell antagonism
Gut
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Hepatocyte dedifferentiation profiling in alcohol-related liver disease identifies CXCR4 as a driver of cell reprogramming
Journal of Hepatology, Vol. 79, Núm. 3, pp. 728-740
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Neuroblastoma RAS viral oncogene homolog (N-RAS) deficiency aggravates liver injury and fibrosis
Cell death & disease, Vol. 14, Núm. 8, pp. 514
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The outcome of boosting mitochondrial activity in alcohol-associated liver disease is organ-dependent
Hepatology (Baltimore, Md.), Vol. 78, Núm. 3, pp. 878-895
2022
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Activation of the unfolded protein response (Upr) is associated with cholangiocellular injury, fibrosis and carcinogenesis in an experimental model of fibropolycystic liver disease
Cancers, Vol. 14, Núm. 1
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Genetic and pharmacological inhibition of XBP1 protects against APAP hepatotoxicity through the activation of autophagy
Cell death & disease, Vol. 13, Núm. 2, pp. 143
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New molecular mechanisms in cholangiocarcinoma: signals triggering interleukin-6 production in tumor cells and KRAS co-opted epigenetic mediators driving metabolic reprogramming
Journal of Experimental and Clinical Cancer Research, Vol. 41, Núm. 1
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Next-generation sequencing of bile cell-free DNA for the early detection of patients with malignant biliary strictures
Gut, Vol. 71, Núm. 6, pp. 1141-1151
2021
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Abnormal liver function test in patients infected with coronavirus (Sars-cov-2): A retrospective single-center study from spain
Journal of Clinical Medicine, Vol. 10, Núm. 5, pp. 1-18
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An Experimental DUAL Model of Advanced Liver Damage
Hepatology Communications, Vol. 5, Núm. 6, pp. 1051-1068
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Dual Targeting of G9a and DNA Methyltransferase-1 for the Treatment of Experimental Cholangiocarcinoma
Hepatology, Vol. 73, Núm. 6, pp. 2380-2396
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Fibrotic events in the progression of cholestatic liver disease
Cells, Vol. 10, Núm. 5
2020
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Loss of c-Jun N-terminal Kinase 1 and 2 Function in Liver Epithelial Cells Triggers Biliary Hyperproliferation Resembling Cholangiocarcinoma
Hepatology Communications, Vol. 4, Núm. 6, pp. 834-851
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Pilot multi-omic analysis of human bile from benign and malignant biliary strictures: A machine-learning approach
Cancers, Vol. 12, Núm. 6, pp. 1-30
2019
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Mitogen-Activated Protein Kinases (MAPKs) and Cholangiocarcinoma: The Missing Link
Cells, Vol. 8, Núm. 10