(FM) Patología, Anatomía y Fisiología
Departamento académico
Stanford University School of Medicine
Stanford, Estados UnidosPublicacións en colaboración con investigadores/as de Stanford University School of Medicine (15)
2024
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GeNNius: an ultrafast drug-target interaction inference method based on graph neural networks
Bioinformatics (Oxford, England), Vol. 40, Núm. 1
2023
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LAMC2 Regulates Key Transcriptional and Targetable Effectors to Support Pancreatic Cancer Growth
Clinical cancer research : an official journal of the American Association for Cancer Research, Vol. 29, Núm. 6, pp. 1137-1154
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Signature-driven repurposing of Midostaurin for combination with MEK1/2 and KRASG12C inhibitors in lung cancer
Nature Communications, Vol. 14, Núm. 1
2021
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Elevated NSD3 histone methylation activity drives squamous cell lung cancer
Nature, Vol. 590, Núm. 7846, pp. 504-508
2020
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The Mir181ab1 cluster promotes KRAS-driven oncogenesis and progression in lung and pancreas
Journal of Clinical Investigation, Vol. 130, Núm. 4, pp. 1879-1895
2019
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Antitumor activity of an engineered decoy receptor targeting CLCF1–CNTFR signaling in lung adenocarcinoma
Nature Medicine, Vol. 25, Núm. 11, pp. 1783-1795
2017
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An integrative approach unveils FOSL1 as an oncogene vulnerability in KRAS-driven lung and pancreatic cancer
Nature Communications, Vol. 8
2016
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The present and the future of the RAS pathway: From function and genomics to inhibition
Cancer Biology and Therapy, Vol. 17, Núm. 7, pp. 719-722
2015
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Analysis of heritability and shared heritability based on genome-wide association studies for thirteen cancer types
Journal of the National Cancer Institute, Vol. 107, Núm. 12
2014
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A meta-Analysis of lung cancer gene expression identifies PTK7 as a survival gene in lung adenocarcinoma
Cancer Research, Vol. 74, Núm. 10, pp. 2892-2902
2012
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Cross-species functional analysis of cancer-associated fibroblasts identifies a critical role for CLCF1 and IL-6 in non-small cell lung cancer in vivo
Cancer Research, Vol. 72, Núm. 22, pp. 5744-5756
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Twist1 suppresses senescence programs and thereby accelerates and maintains mutant Kras-induced lung tumorigenesis
PLoS Genetics, Vol. 8, Núm. 5
2011
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Tumor-stromal interactions of the bone microenvironment: In vitro findings and potential in vivo relevance in metastatic lung cancer models
Clinical and Experimental Metastasis, Vol. 28, Núm. 8, pp. 779-791
2010
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Wilms tumor 1 (WT1) regulates KRAS-driven oncogenesis and senescence in mouse and human models
Journal of Clinical Investigation, Vol. 120, Núm. 11, pp. 3940-3952
2008
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A novel lung cancer signature mediates metastatic bone colonization by a dual mechanism
Cancer Research, Vol. 68, Núm. 7, pp. 2275-2285