Publicaciones en las que colabora con Rafael Franco Fernandez (20)

2022

  1. Genetic Inactivation of Free Fatty Acid Receptor 3 Impedes Behavioral Deficits and Pathological Hallmarks in the APPswe Alzheimer’s Disease Mouse Model

    International Journal of Molecular Sciences, Vol. 23, Núm. 7

2019

  1. Dopamine receptor D3 expression is altered in CD4+ T-cells from Parkinson's disease patients and its pharmacologic inhibition attenuates the motor impairment in a mouse model

    Frontiers in Immunology, Vol. 10, Núm. MAY

2018

  1. Maternal imprinting on cognition markers of wild type and transgenic Alzheimer's disease model mice

    Scientific Reports, Vol. 8, Núm. 1

2017

  1. GPR55: A therapeutic target for Parkinson's disease?

    Neuropharmacology, Vol. 125, pp. 319-332

  2. Pharmacologic antagonism of dopamine receptor D3 attenuates neurodegeneration and motor impairment in a mouse model of Parkinson's disease

    Neuropharmacology, Vol. 113, pp. 110-123

2016

  1. Fatty acid amide hydrolase inhibition for the symptomatic relief of Parkinson's disease

    Brain, Behavior, and Immunity, Vol. 57, pp. 94-105

  2. Neuroprotective Effect of JZL184 in MPP+-Treated SH-SY5Y Cells Through CB2 Receptors

    Molecular Neurobiology, Vol. 53, Núm. 4, pp. 2312-2319

2015

  1. Structures for G-Protein-Coupled Receptor Tetramers in Complex with G Proteins

    Trends in Biochemical Sciences

2014

  1. GPR40 activation leads to CREB and ERK phosphorylation in primary cultures of neurons from the mouse CNS and in human neuroblastoma cells

    Hippocampus, Vol. 24, Núm. 7, pp. 733-739

  2. The monoacylglycerol lipase inhibitor JZL184 is neuroprotective and alters glial cell phenotype in the chronic MPTP mouse model

    Neurobiology of Aging, Vol. 35, Núm. 11, pp. 2603-2616

2013

  1. Age-related mitochondrial alterations without neuronal loss in the hippocampus of a transgenic model of Alzheimer's disease

    Current Alzheimer Research, Vol. 10, Núm. 4, pp. 390-405

  2. Phenyl Acyl Acids Attenuate the Unfolded Protein Response in Tunicamycin-Treated Neuroblastoma Cells

    PLoS ONE, Vol. 8, Núm. 8

2012

  1. Chronic mild stress accelerates the onset and progression of the Alzheimer's disease phenotype in Tg2576 mice

    Journal of Alzheimer's Disease, Vol. 28, Núm. 3, pp. 567-578

  2. Loss of parvalbumin-positive neurons from the globus pallidus in animal models of parkinson disease

    Journal of Neuropathology and Experimental Neurology, Vol. 71, Núm. 11, pp. 973-982

2011

  1. Chronic mild stress in mice promotes cognitive impairment and CDK5-dependent tau hyperphosphorylation

    Behavioural Brain Research, Vol. 220, Núm. 2, pp. 338-343

  2. Real-time G-protein-coupled receptor imaging to understand and quantify receptor dynamics

    TheScientificWorldJournal, Vol. 11, pp. 1995-2010

2010

  1. Calcium-mediated modulation of the quaternary structure and function of adenosine A2A-dopamine D2 receptor heteromers

    Current Opinion in Pharmacology, Vol. 10, Núm. 1, pp. 67-72

  2. Cocaine self-administration markedly increases dopamine D2 receptor negative cooperativity for dopamine binding: A receptor dimer-based analysis

    Synapse, Vol. 64, Núm. 7, pp. 566-569

  3. Direct involvement of σ-1 receptors in the dopamine D1 receptor-mediated effects of cocaine

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 107, Núm. 43, pp. 18676-18681

2009

  1. Interactions between calmodulin, adenosine A2A, and dopamine D2 receptors

    Journal of Biological Chemistry, Vol. 284, Núm. 41, pp. 28058-28068