(FC) Bioquímica y Genética
Departamento académico
Miguel Ángel
Abellanas Sánchez
Publicaciones en las que colabora con Miguel Ángel Abellanas Sánchez (11)
2024
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Nigrostriatal degeneration determines dynamics of glial inflammatory and phagocytic activity
Journal of Neuroinflammation, Vol. 21, Núm. 1
2023
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Microglia and astrocyte activation is region-dependent in the α-synuclein mouse model of Parkinson's disease
GLIA, Vol. 71, Núm. 3, pp. 571-587
2021
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Expression of endothelial nox5 alters the integrity of the blood-brain barrier and causes loss of memory in aging mice
Antioxidants, Vol. 10, Núm. 8
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Gut microbial dysbiosis after traumatic brain injury modulates the immune response and impairs neurogenesis
Acta Neuropathologica Communications, Vol. 9, Núm. 1
2020
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Cannabinoid receptor type 2 as a therapeutic target for Parkinson’s disease
Diagnosis and Management in Parkinson’s Disease: The Neuroscience of Parkinson’s Disease, Volume 1 (Elsevier), pp. 557-573
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Cb2 receptors and neuron–glia interactions modulate neurotoxicity generated by magl inhibition
Biomolecules, Vol. 10, Núm. 8, pp. 1-15
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Mecanismos inmunosupresores de la microglía del mesencéfalo en modelos de neuroinflamación y daño neurológico
Mecanismos inmunosupresores de la microglía del mesencéfalo en modelos de neuroinflamación y daño neurológico
2019
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Dopamine receptor D3 expression is altered in CD4+ T-cells from Parkinson's disease patients and its pharmacologic inhibition attenuates the motor impairment in a mouse model
Frontiers in Immunology, Vol. 10, Núm. MAY
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Midbrain microglia mediate a specific immunosuppressive response under inflammatory conditions
Journal of Neuroinflammation, Vol. 16, Núm. 1
2018
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Cannabinoid pharmacology/therapeutics in chronic degenerative disorders affecting the central nervous system
Biochemical Pharmacology, Vol. 157, pp. 67-84
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The expression of cannabinoid type 1 receptor and 2-arachidonoyl glycerol synthesizing/degrading enzymes is altered in basal ganglia during the active phase of levodopa-induced dyskinesia
Neurobiology of Disease, Vol. 118, pp. 64-75