(FC) Bioquímica y Genética
Departamento académico
Icahn School of Medicine at Mount Sinai
Nueva York, Estados UnidosPublicaciones en colaboración con investigadores/as de Icahn School of Medicine at Mount Sinai (16)
2024
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Integrating Multi-Omics with environmental data for precision health: A novel analytic framework and case study on prenatal mercury induced childhood fatty liver disease
Environment International, Vol. 190
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Letter to the Editor: Serum Identification of At-Risk MASH: The Metabolomics-Advanced Steatohepatitis Fibrosis Score (MASEF)
Hepatology
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SUMOylation controls Hu antigen R posttranscriptional activity in liver cancer
Cell Reports, Vol. 43, Núm. 3
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Serum identification of at-risk MASH: The metabolomics-advanced steatohepatitis fibrosis score (MASEF)
Hepatology, Vol. 79, Núm. 1, pp. 135-148
2023
2022
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Activation of the unfolded protein response (Upr) is associated with cholangiocellular injury, fibrosis and carcinogenesis in an experimental model of fibropolycystic liver disease
Cancers, Vol. 14, Núm. 1
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Association of Prenatal Exposure to Endocrine-Disrupting Chemicals with Liver Injury in Children
JAMA Network Open, Vol. 5, Núm. 7, pp. E2220176
2021
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In Utero Exposure to Mercury Is Associated With Increased Susceptibility to Liver Injury and Inflammation in Childhood
Hepatology, Vol. 74, Núm. 3, pp. 1546-1559
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Mapping the human genetic architecture of COVID-19
Nature, Vol. 600, Núm. 7889, pp. 472-477
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Prenatal and childhood exposure to air pollution and traffic and the risk of liver injury in European children
Environmental Epidemiology, Vol. 5, Núm. 3
2020
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Prenatal Exposure to Perfluoroalkyl Substances Associated With Increased Susceptibility to Liver Injury in Children
Hepatology, Vol. 72, Núm. 5, pp. 1758-1770
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Reversal of Object Recognition Memory Deficit in Perirhinal Cortex-Lesioned Rats and Primates and in Rodent Models of Aging and Alzheimer's Diseases
Neuroscience, Vol. 448, pp. 287-298
2017
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Global position paper on cardiovascular regenerative medicine
European Heart Journal
2013
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GCN2 kinase is a key regulator of fibrogenesis and acute and chronic liver injury induced by carbon tetrachloride in mice
Laboratory Investigation, Vol. 93, Núm. 3, pp. 303-310
2000
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Tumor necrosis factor α down-regulates expression of the α1(I) collagen gene in rat hepatic stellate cells through a p20C/EBPβ- and C/EBPδ-dependent mechanism
Hepatology, Vol. 31, Núm. 5, pp. 1086-1093