Publicacions en què col·labora amb María Esther Larrea Leoz (20)

2014

  1. Characterization of the CD40L/Oncostatin M/Oncostatin M receptor axis as an antiviral and immunostimulatory system disrupted in chronic HCV infection

    Journal of Hepatology, Vol. 60, Núm. 3, pp. 482-489

  2. Dysregulation of interferon regulatory factors impairs the expression of immunostimulatory molecules in hepatitis C virus genotype 1-infected hepatocytes

    Gut, Vol. 63, Núm. 4, pp. 665-673

2008

  1. Implication of human N-α-acetyltransferase 5 in cellular proliferation and carcinogenesis

    Oncogene, Vol. 27, Núm. 58, pp. 7296-7306

2007

  1. HCV structural proteins interfere with interferon-alpha Jak/STAT signalling pathway

    Antiviral Research, Vol. 76, Núm. 2, pp. 194-197

  2. Upregulation of indoleamine 2,3-dioxygenase in hepatitis C virus infection

    Journal of Virology, Vol. 81, Núm. 7, pp. 3662-3666

2006

  1. Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: In vivo and in vitro studies

    Gut, Vol. 55, Núm. 8, pp. 1188-1196

  2. Recombinant Adenoviral Vectors Turn on the Type I Interferon System without Inhibition of Transgene Expression and Viral Replication

    Molecular Therapy, Vol. 14, Núm. 1, pp. 129-138

2005

  1. Hepatitis C virus infection of primary tupaia hepatocytes leads to selection of quasispecies variants, induction of interferon-stimulated genes and NF-κB nuclear translocation

    Journal of General Virology, Vol. 86, Núm. 11, pp. 3065-3074

2004

  1. IFN-α5 mediates stronger Tyk2-stat-dependent activation and higher expression of 2′,5′-oligoadenylate synthetase than IFN-α2 in liver cells

    Journal of Interferon and Cytokine Research, Vol. 24, Núm. 8, pp. 497-503

2001

  1. Dendritic cells infected with recombinant defective adenoviruses undergo NF-kB activation and partial maturation

    INMUNOLOGÍA, Vol. 20, Núm. 3, pp. 130-142

  2. Expression of interferon-α subtypes in peripheral mononuclear cells from patients with chronic hepatitis C: A role for interferon-α5

    Journal of Viral Hepatitis, Vol. 8, Núm. 2, pp. 103-110

  3. Nuclear factor-kB in the liver of patients with chronic hepatitis C: Decreased RelA expression is associated with enhanced fibrosis progression

    Hepatology, Vol. 34, Núm. 5, pp. 1041-1048

1999

  1. Antioxidant status and glutathione metabolism in peripheral blood mononuclear cells from patients with chronic hepatitis C

    Journal of Hepatology, Vol. 31, Núm. 5, pp. 808-814

  2. Interferon alfa subtypes and levels of type I interferons in the liver and peripheral mononuclear cells in patients with chronic hepatitis C and controls

    Hepatology, Vol. 29, Núm. 6, pp. 1900-1904

1998

  1. Superoxide dismutase in patients with chronic hepatitis C virus infection

    Free Radical Biology and Medicine, Vol. 24, Núm. 7-8, pp. 1235-1241

1996

  1. Tumor necrosis factor α gene expression and the response to interferon in chronic hepatitis C

    Hepatology, Vol. 23, Núm. 2, pp. 210-217

1994

  1. Enhanced expression of TNFα in patients with primary biliary cirrhosis

    International Hepatology Communications, Vol. 2, Núm. 1, pp. 6-13

  2. Prediction of sustained remission of chronic hepatitis C after a 12-month course of alfa interferon

    Journal of Hepatology, Vol. 21, Núm. 1, pp. 4-11

1992

  1. The stilbene disulfonic acid DIDS stimulates the production of TNF-α in human lymphocytes

    Biochemical and Biophysical Research Communications, Vol. 189, Núm. 3, pp. 1268-1274

  2. Tratamiento de la hepatitis crónica B con interferón alfa linfoblastoide: efectos en la inmunidad celular

    Gastroenterologia y Hepatologia, Vol. 15, Núm. 8, pp. 437-442