Publicaciones en las que colabora con María Pilar Civeira Murillo (23)

2014

  1. Characterization of the CD40L/Oncostatin M/Oncostatin M receptor axis as an antiviral and immunostimulatory system disrupted in chronic HCV infection

    Journal of Hepatology, Vol. 60, Núm. 3, pp. 482-489

  2. Dysregulation of interferon regulatory factors impairs the expression of immunostimulatory molecules in hepatitis C virus genotype 1-infected hepatocytes

    Gut, Vol. 63, Núm. 4, pp. 665-673

2008

  1. Implication of human N-α-acetyltransferase 5 in cellular proliferation and carcinogenesis

    Oncogene, Vol. 27, Núm. 58, pp. 7296-7306

  2. Monocyte-derived dendritic cells from HCV-infected patients transduced with an adenovirus expressing NS3 are functional when stimulated with the TLR3 ligand poly(I:C)

    Journal of Viral Hepatitis, Vol. 15, Núm. 11, pp. 782-789

2007

  1. HCV structural proteins interfere with interferon-alpha Jak/STAT signalling pathway

    Antiviral Research, Vol. 76, Núm. 2, pp. 194-197

  2. Upregulation of indoleamine 2,3-dioxygenase in hepatitis C virus infection

    Journal of Virology, Vol. 81, Núm. 7, pp. 3662-3666

2006

  1. Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: In vivo and in vitro studies

    Gut, Vol. 55, Núm. 8, pp. 1188-1196

  2. Characterization of T-cell responses against immunodominant epitopes from hepatitis C virus E2 and NS4a proteins

    Journal of Viral Hepatitis, Vol. 13, Núm. 1, pp. 47-55

  3. Recombinant Adenoviral Vectors Turn on the Type I Interferon System without Inhibition of Transgene Expression and Viral Replication

    Molecular Therapy, Vol. 14, Núm. 1, pp. 129-138

2005

  1. Hepatitis C virus infection of primary tupaia hepatocytes leads to selection of quasispecies variants, induction of interferon-stimulated genes and NF-κB nuclear translocation

    Journal of General Virology, Vol. 86, Núm. 11, pp. 3065-3074

2004

  1. IFN-α5 mediates stronger Tyk2-stat-dependent activation and higher expression of 2′,5′-oligoadenylate synthetase than IFN-α2 in liver cells

    Journal of Interferon and Cytokine Research, Vol. 24, Núm. 8, pp. 497-503

2001

  1. Dendritic cells infected with recombinant defective adenoviruses undergo NF-kB activation and partial maturation

    INMUNOLOGÍA, Vol. 20, Núm. 3, pp. 130-142

2000

  1. Pathological and virological findings in patients with persistent hypertransaminasaemia of unknown aetiology

    Gut, Vol. 47, Núm. 3, pp. 429-435

1999

  1. Immunogenicity of variable regions of hepatitis C virus proteins: Selection and modification of peptide epitopes to assess hepatitis C virus genotypes by ELISA

    Journal of General Virology, Vol. 80, Núm. 3, pp. 727-738

1998

  1. Cellular immunity to hepatitis C virus core protein and the response to interferon in patients with chronic hepatitis C

    Hepatology, Vol. 28, Núm. 3, pp. 815-822

  2. Transmission of hepatitis C virus infection to tree shrews

    Virology, Vol. 244, Núm. 2, pp. 513-520

1997

  1. Viremia after one month of interferon therapy predicts treatment outcome in patients with chronic hepatitis C

    Gastroenterology, Vol. 113, Núm. 5, pp. 1647-1653

1996

  1. Further insights on the inhibition of HIV type 1 infection in vitro by CD4-modified synthetic peptides containing phenylalanine

    AIDS Research and Human Retroviruses, Vol. 12, Núm. 11, pp. 1023-1030

  2. Production of interleukin-2 in response to synthetic peptides from hepatitis C virus E1 protein in patients with chronic hepatitis C: Relationship with the response to interferon treatment

    Journal of Hepatology, Vol. 25, Núm. 1, pp. 1-9

1995

  1. Interferon treatment of chronic hepatitis C: a randomised, controlled study of human lymphoblastoid alpha-interferon versus no therapy

    Lymphoblastoid alpha-interferon (Current Medical Literature Ltd), pp. 113 - 122