Facultad de Medicina (FM)
Centro académico
Jesús María
Prieto Valtueña
Investigador ata 2014
Publicacións nas que colabora con Jesús María Prieto Valtueña (375)
2023
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Application of a split-Cre system for high-capacity adenoviral vector amplification
Biotechnology Journal, Vol. 18, Núm. 3
2022
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Recombinant porphobilinogen deaminase targeted to the liver corrects enzymopenia in a mouse model of acute intermittent porphyria
Science Translational Medicine, Vol. 14, Núm. 627
2020
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Brain ventricular enlargement in human and murine acute intermittent porphyria
Human molecular genetics, Vol. 29, Núm. 19, pp. 3211-3223
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Disruption of SIRT7 Increases the Efficacy of Checkpoint Inhibitor via MEF2D Regulation of Programmed Cell Death 1 Ligand 1 in Hepatocellular Carcinoma Cells
Gastroenterology, Vol. 158, Núm. 3, pp. 664-678.e24
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Identifying differentially expressed micrornas, target genes, and key pathways deregulated in patients with liver diseases
International Journal of Molecular Sciences, Vol. 21, Núm. 19, pp. 1-16
2019
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Acute Intermittent Porphyria: Novel Etiologic and Pathogenic Therapies Based on RNA Transfer to the Liver
Hepatology
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Cardiotrophin-1 is an anti-inflammatory cytokine and promotes IL-4–induced M2 macrophage polarization
FASEB Journal, Vol. 33, Núm. 6, pp. 7578-7587
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La clínica y el laboratorio: interpretación de análisis y pruebas funcionales, exploración de los síndromes, cuadro biológico de las enfermedades
Elsevier
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Liver Expression of a MiniATP7B Gene Results in Long-Term Restoration of Copper Homeostasis in a Wilson Disease Model in Mice
Hepatology, Vol. 70, Núm. 1, pp. 108-126
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Short-Term Local Expression of a PD-L1 Blocking Antibody from a Self-Replicating RNA Vector Induces Potent Antitumor Responses
Molecular Therapy, Vol. 27, Núm. 11, pp. 1892-1905
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The cirrhotic liver is depleted of docosahexaenoic acid (DHA), a key modulator of NF-κB and TGFβ pathways in hepatic stellate cells
Cell Death and Disease, Vol. 10, Núm. 1
2018
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Bile acids, FGF15/19 and liver regeneration: From mechanisms to clinical applications
Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 4, pp. 1326-1334
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Erratum to “A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction” [J Hepatol 67 (2017) 669–679] (S0168827817320469) (10.1016/j.jhep.2017.05.010))
Journal of Hepatology
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TMEM173 Alternative Spliced Isoforms Modulate Viral Replication through the STING Pathway
ImmunoHorizons, Vol. 2, Núm. 11, pp. 363-376
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Targeting the anion exchanger 2 with specific peptides as a new therapeutic approach in b lymphoid neoplasms
Haematologica, Vol. 103, Núm. 6, pp. 1065-1072
2017
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A new HDV mouse model identifies mitochondrial antiviral signaling protein (MAVS) as a key player in IFN-β induction
Journal of Hepatology, Vol. 67, Núm. 4, pp. 669-679
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Blockage of FOXP3 transcription factor dimerization and FOXP3/AML1 interaction inhibits T regulatory cell activity: sequence optimization of a peptide inhibitor
Oncotarget, Vol. 8, Núm. 42, pp. 71709-71724
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Engineered fibroblast growth factor 19 protects from acetaminophen-induced liver injury and stimulates aged liver regeneration in mice
Cell Death and Disease, Vol. 8, Núm. 10
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Fibroblast growth factor 15/19 (FGF15/19) protects from diet-induced hepatic steatosis: Development of an FGF19-based chimeric molecule to promote fatty liver regeneration
Gut, Vol. 66, Núm. 10, pp. 1818-1828
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Improvement of Adeno-Associated Virus-Mediated Liver Transduction Efficacy by Regional Administration in Macaca fascicularis
Human Gene Therapy Clinical Development, Vol. 28, Núm. 2, pp. 68-73